新冠肺炎|法醫病理學教授劉良:針對缺氧患者「單純給氧氣」,恐有反作用

中國大陸華中科技大學同濟醫學院的法醫病理學教授劉良,帶領團隊在2月16日完成了第一例新冠肺炎死亡病例的屍體解剖,試圖找出武漢肺炎對身體造成的損傷!

 

劉良提出「肯定不止損害肺,還包括免疫系統和身體其他器官」,而且因為武漢肺炎的特殊性,現階段的治療方式,如果針對缺氧的患者「單純給氧氣」,反而會讓患者肺部堵塞的更嚴重,更容易缺氧。

 

中國國家工程院院士鍾南山說過,武漢肺炎患者的遺體解剖後,可以發現肺中有大量的黏液。劉良也表示,「我們在顯微鏡下,確實發現有些死者肺部切面上有黏液性的分泌物,這可以提醒臨床治療提高警惕。」

 

肺泡功能好,才可以確保人體不窒息。武漢肺炎可能損傷肺泡的功能,加上呼吸道又被黏液堵住,臨床上才會出現缺氧的表現。

 

劉良提出「要改善人的缺氧狀態,保持氣道通暢,必須對黏液進行稀釋或者溶解,實際上目前已經使用了這類藥物,但可以再多試一試。但如果黏液成分沒有化解,單純用給氧的方式,可能達不到目的,有時候會起反作用正壓給氧的時候可能會把黏液推得更深更廣,會加重患者的缺氧。」

 

所以針對重症患者的治療,單純給予呼吸器、或是心肺功能機可能不是解藥,要同時時解決「肺部黏液」太多的問題。

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Margaret W. Lavigne 司馬儀

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